![]() 30) It is mainly caused by minor trauma, and nonunion occurs due to avascular necrosis at the vertebral body fracture site, which can lead to vertebral kyphosis or intravertebral instability. Kummell’s disease (KD) was first reported by Hermann Kummell in 1895 and is called delayed posttraumtic vertebral collapse, avascular necrosis of vertebrae, or ischemic vertebral collapse. We have presented a literature review and discussed clinical guidelines and therapeutic strategies to reduce the morbidity and mortality associated with KD. In this study, we summarize the pathogenesis, diagnosis, and treatment of KD, which is showing increasing prevalence in an aging society. Timely interventions such as vertebral augmentation or surgery, with active regular follow-up are necessary before the onset of neurological deficits due to osteonecrotic collapse in patients with suspected KD. KD is considered a complication of osteoporotic vertebral compression fractures, and conservative treatment, including osteoporosis treatment, is important. Clinical symptoms of KD range from no symptoms to severe paralysis due to nerve injury. It is mainly caused by minor trauma, and nonunion occurs secondary to avascular necrosis at the vertebral body fracture site, which can lead to vertebral kyphosis or intravertebral instability. KD is no longer rare in an aging society. Kummell’s disease (KD) is referred to as delayed posttraumatic vertebral collapse, avascular necrosis of vertebrae, or ischemic vertebral collapse.
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